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In calcific tendinitis of the shoulder (tendinosis calcarea), calcium is deposited in a tendon of the rotator cuff — the cap of muscle and tendon that guides the head of your upper arm bone in its socket. The tendon involved is usually the supraspinatus tendon, which runs beneath the roof of the shoulder. The deposit itself is neither an inflammation nor a fragment of bone, but a chalk-like material within the tendon tissue that can at times have almost the consistency of toothpaste.¹
One point is key to understanding it: calcium in the tendon does not automatically mean pain. Deposits are often picked up by chance on X-rays taken for entirely different reasons — in people who have never had any symptoms. The typical, often severe pain only appears once the calcium narrows the gliding space beneath the roof of the shoulder, or once the body starts to break it down.
Calcific tendinitis has to be told apart from other causes of shoulder pain, above all from impingement syndrome, from a rotator cuff tear and from osteoarthritis of the shoulder joint. That distinction is usually straightforward, because the deposit shows up directly on an X-ray.
According to the classic model described by Uhthoff, calcific tendinitis passes through several stages. This model explains why symptoms can simmer for months and then suddenly escalate — and why patience is a genuine treatment strategy.¹,²
The good news in all of this: the worst phase is precisely the one in which the body is already solving the problem itself. That is why surgery is usually not the first step — instead the pain is bridged until the breakdown is complete. How long that takes cannot be predicted in an individual case; courses lasting many months to years are possible.
The most relevant consequence is not the deposit itself but the loss of movement caused by protecting the joint. If the shoulder is barely moved for weeks, the joint capsule can contract — up to a frozen shoulder, which takes considerably longer to settle than the calcific tendinitis itself. That is why early, pain-adapted movement belongs in every treatment plan. Sleep problems caused by night pain are a serious issue too, because they further amplify how pain is perceived.
Why calcium forms in a tendon of all places is, according to current knowledge, not fully understood. The main factors under discussion are the local blood supply and remodelling processes in the tendon tissue. The following count as risk factors:²,³
The diagnosis is usually uncomplicated, because imaging shows the deposit directly. A typical sequence looks like this:
On the X-ray the deposit is often graded according to Gärtner: sharply outlined, dense deposits suggest a quiet phase, while cloudy, ill-defined deposits suggest that resorption is already under way. This grading matters in practice because it shapes the choice of treatment — a deposit that is currently dissolving calls for pain relief and time rather than shockwave therapy.²
Treatment follows the phase and the burden of symptoms, not the size of the deposit. As a rule it proceeds in steps — conservative measures first, invasive procedures only once months have passed without improvement. The decision is always made by your treating practice together with you.
Medicines treat the pain and the accompanying inflammation — they do not dissolve the calcium deposit. That is precisely why they are meant as a bridge and not as a permanent solution.
Non-steroidal anti-inflammatory drugs such as ibuprofen, diclofenac or naproxen are the option of choice in the acute phase. Naproxen lasts longer and is therefore often preferred where night pain is the problem. To weigh up the choice in an individual case, it is worth a look at our painkiller comparison.
Where NSAIDs are not an option, paracetamol or metamizole are used instead. Neither has an anti-inflammatory effect, but both can dampen the pain sufficiently. Metamizole is prescription-only in Germany and calls for attention to rare but serious changes in the blood count.
Injected beneath the roof of the shoulder, prednisolone or a comparable glucocorticoid can quickly calm an acute flare of irritation. As a tablet, corticosteroids are not usually given long term for calcific tendinitis. The general picture is set out in our cortisone guide.
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Plenty of promises circulate around calcific tendinitis. Here is a sober look at the three procedures that come up most often.
| Procedure | Principle | Suitable for | Assessment |
|---|---|---|---|
| Shockwave (ESWT) | Energy waves applied to the deposit from outside | Rather dense, quiet deposits; several sessions | May ease symptoms; according to current evidence the effect is moderate and not present in everyone; in Germany usually a self-pay service |
| Needling / lavage | Ultrasound-guided puncture and flushing | Soft deposits in the resorption phase | May shorten the course; a short-term increase in pain after the procedure is possible |
| Arthroscopy | Surgical removal of the deposit | Long-standing cases that resist treatment | Good results in selected cases, but an operation with aftercare — hence late in the stepped plan |
One thing that keeps standing out in studies: many cases improve even without any of these procedures. That makes individual methods hard to judge — and at the same time it is the strongest argument for a watchful first step.
The last point in particular rewards consistency: many people take NSAIDs “as needed” for weeks and underestimate the total amount. How to keep a clean record is set out in How to take medications and, once several products come together, in Drug interactions.
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