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Medically reviewed guide · Last updated: 1 August 2026 · Reading time: approx. 9 min
At a glance
| Phenomenon | What it looks like | How long | Typical accompanying signs | First step |
|---|---|---|---|---|
| Scintillating scotoma (migraine aura) | Zigzag or jagged pattern, often with a blind spot, travels slowly outwards across your field of vision | 20–30 minutes, then completely gone | Often a headache afterwards, sensitivity to light, nausea | Rest, avoid stimulation; have it assessed medically the first time it happens |
| Floaters, Mouches volantes (vitreous opacities) | Floating threads, dots or rings that move with your gaze and "drift" after it | Permanently present, sometimes more and sometimes less noticeable | Clearest against a bright background, no pain | Usually harmless; see an eye specialist if they suddenly increase markedly |
| Flashes of light (photopsia) | Brief flashes at the edge of your field of vision, often on eye movement or in the dark | Fractions of a second, repeatedly | Frequently together with new black spots | New in onset: have it assessed by an eye specialist the same day |
| Circulation and blood sugar | Diffuse shimmering or a "snow flurry" in both eyes, often with things going black | Seconds to a few minutes | Standing up, heat, hunger, trembling, sweating | Sit down, check your blood sugar, discuss your blood pressure medication |
| Strain and irritation | Slight flickering, blurring, sensitivity to glare | Minutes to hours, worse in the evening | Dry eyes, screen work, lack of sleep, a lot of caffeine | Breaks, artificial tears, sleep — if it does not improve, get it checked |
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The scintillating scotoma is the best-known form of visual aura in migraine. It usually begins as a small shimmering point near the centre of your gaze, grows over minutes into a jagged arc and travels slowly outwards; in the middle of the pattern there is often nothing to see at all — hence the term scotoma (a gap in the field of vision). After 20 to 30 minutes as a rule the aura disappears completely, and for many people the typical one-sided headache then follows.
What matters for making sense of it: an aura travels and builds up. A visual problem that is fully there from one second to the next and then stays does not fit an aura — an important difference from a circulatory disturbance. Because the aura arises in the visual centre of the brain and not in the eye, it is visible in both eyes.
Some people experience an aura without a headache afterwards. That happens and is not in itself threatening. To be honest, though, one rule holds: if an aura like this appears for the first time later in life — beyond the age of 50, say — with no history of migraine at all, it should be assessed medically. Temporary circulatory disturbances in the brain cannot be told apart from an aura on the description alone.
Over the course of life the gel inside the eye liquefies and individual fibres clump together. These clumps cast a shadow on the retina — visible as grey threads, dots or "midges" that swim along when you move your gaze and drift on slightly when you hold still. They are clearest against bright surfaces: a white wall, snow, the sky, a screen.
As a rule they are harmless, they are more common in short-sightedness and they increase with age; the brain usually learns to filter them out well. For the harmless form there is essentially no treatment — surgical procedures on the vitreous carry risks of their own. The point at which it tips over: not the threads that are already there, but a sudden marked increase within hours.
The retina has no sensation of its own for touch — it reports every stimulus as light. So when the shrinking vitreous pulls on it, the impression is of brief flashes, typically at the edge of the field of vision, often on eye movement or in the dark. This posterior vitreous detachment is a normal part of ageing and usually unproblematic.
It can, however, adhere so firmly in one place that it tears the retina. A retinal tear like this is the precursor of a retinal detachment — an ophthalmic emergency, because lasting eyesight is at stake. So with newly appeared flashes the rule is: do not wait, but have the back of the eye examined promptly with the pupil dilated.
Shimmering or a "snow flurry" in both eyes when you get up quickly, in a sauna or after standing for a long time is a classic circulatory phenomenon: blood pressure drops briefly and the retina is less well supplied for a few seconds. If that happens to you often, read the explanation under low blood pressure — and see whether it fits in time with new or increased blood pressure medication. A hypo is just as typical: shimmering, blurred vision, trembling, sweating and ravenous hunger often occur together. Anyone who has diabetes and takes active substances that actively lower blood sugar should always read flickering vision as a possible sign of a hypo as well.
An acute rise in the pressure inside the eye — an acute angle-closure attack in glaucoma — can cause coloured rings around lights, misty vision, a red eye, severe pain and nausea; an emergency that feels distinctly different from harmless shimmering. Swings in blood sugar also change the refractive power of the lens, which is perceived as blurred vision and flickering. Anything that changes your sight lastingly belongs in the assessment set out under visual disturbances.
A great deal of flickering vision has no underlying condition as its cause, simply overload: screen work with a reduced blink rate, dry eyes, too little sleep, not drinking enough, skipped meals, a lot of caffeine or caffeine withdrawal. Typically it increases towards the evening and improves with breaks. If the flickering persists all the same, that is a finding in itself.
These observations do not replace a diagnosis. But in a few minutes they set the key points, and they make the conversation at your appointment much more precise.
Without emergency signs, the approach is usually to work from the simple to the more elaborate.
This section is the one most often skipped over with flickering vision — and it is often the most productive. In that case the eye is not what is unwell: the supply is fluctuating, or an active substance is acting directly on the retina.
Anyone treated for high blood pressure knows the goal "lower is better" — until blood pressure drops too far on standing. What is typical then is shimmering or things going black in front of your eyes when you get up, in the morning after the first tablet or on hot days. The trigger is often an increase in dose, a new combination, or diuretics when you are not drinking enough. That is no reason to stop medicines on your own initiative — but a good reason to document how things go and to have the dose reviewed.
Active substances that actively lower blood sugar can also lower it too far — among them insulin and sulfonylureas such as glimepiride. Flickering vision, blurred vision, trembling, sweating and ravenous hunger are then not an eye problem but a blood sugar problem. Particularly treacherous: skipped meals, unaccustomed exertion and alcohol. If flickering regularly happens to you before meals, that is the moment to measure your blood sugar.
Cardiac glycosides such as digoxin have a narrow margin between an effective dose and one that is too high. Visual disturbances are a well-known sign of an overdose: flickering, sensitivity to glare, haloes around lamps and a yellow-green tinge to colours, frequently with nausea, loss of appetite and heart rhythm disturbances. This is encouraged by declining kidney function, diuretics and a lack of potassium. New visual disturbances while taking a cardiac glycoside should be reported promptly to your doctor — here the blood level is measured rather than waited out.
Sildenafil inhibits, alongside its target enzyme, a related enzyme in the retina to a small degree. That produces the well-known and usually temporary side effect: a blue or blue-green tinge, increased sensitivity to glare and slight flickering a few hours after taking it, which fades again along with the concentration of the active substance. What is not harmless is a sudden loss of vision in one eye — that needs to be assessed immediately.
Other triggers: products with anticholinergic properties, some antiepileptics at too high a dose, and antimalarial and antirheumatic medicines on long-term use — with those, eye checks are part of the treatment.
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This article is for general information and does not replace medical advice, diagnosis or treatment. The self-observations described — including the cover test — are a guide and not a diagnosis. If you have newly appeared flashes of light with a swarm of dark spots, a shadow in your field of vision, a sudden loss of vision, or visual disturbances together with weakness, altered sensation or difficulty speaking, please contact a doctor or the emergency services without delay.